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Unexplained prolunged fever, anorexia, obesity
To correctly understand this heading:
Remember that any stressed person who continuously clenches his teeth can elicite remote pain and associated dyscomforts from teeth (which are not only painful organs but also tactile one like the thumb and the index in opposite situation)
If You are interested the scientifical data are shown lower.
Don't forget to consult the associated headlings : "Clinical Data" and "Witness and experience"
POSSIBLE UNEXPLAINED PROLUNGED FEVER
& TROUBLES in ALIMENTARY COMPORTMENT
(excess or lack of appetite)
Important research about non-algic inputs that may come from teeth is surprising.
Keep in mind that a stressed person’s teeth, when clenched too tightly or for too long, will send harmful information to a small gland located in the brain: the hypothalamus. It is scientifically proven that teeth send information to the hypothalamus. In some cases the information ends up in the ventromedian nucleus, the center of feeding behavior, which may then be disturbed by the excess information – a situation that leads to what doctors refer to as bulimia. Bulimia can lead to obesity. Stopping jaw clenching and grinding of teeth can lead to weight loss. The following testimony describes the onset and cessation of weight gain. What’s more, teeth send information (again the fact is scientifically proven) to the center of feeding behavior. Stopping the jaw and teeth clenching may explain weight loss, and may also explain a lack of appetite. See also (ANOREXIA).
NEVILLEN H. GOLDEN, I.R. SHENKER (1994) Amenorrhea in anorexia nervosa neuroendocrine control of hypothalamic dysfunction
Amenorrhea is one of the cardinal features of anorexia nervosa and is associated with hypothalamic dysfunction Earlier theories of weight loss, decreased body fat, or exercise do not fully explain the etiology of amenorrhea in anorexia nervosa Disturbances in central dopaminergic and opioid activity have been described in anorexia nervosa and both these substances are known to modulate gonadotropin-releasing hormone (GnRH)-mediated luteinizing hormone (LH) release. Serum LH, follicle stimulating hormone (FSH), estradiol, and prolactin levels were measured at baseline and after administration of metoclopramide (a central D-2 dopamine receptor blocker) in 10 newly diagnosed women with anorexia nervosa and in 10 healthy age-matched Econtrols. Basal prolactin levels and the prolactin response to metoclopramide were significantly impaired in the group with anorexia nervosa. Metoclopramide did not induce a significant rise in LH levels in either the anorexic or the control groups. Neurotransmitter abnormalities may influence hypothalamic dysfunction in anorexia nervosa but the exact mechanism remains to be determined. © by John Wiley & Sons, Inc.
Bruno LEBRUN, Bruno BARIOHAY and André JEAN (2006) CONTROL OF FOOD INTAKE: NEUROBIOLOGICAL ASPECTS Bull. Acad. Vét. France —- Tome 159 - N°4 www.academie-veterinaire-france.fr 289
Neural networks located in the hypothalamic nuclei and the dorsal vagal complex in the brainstem play a crucial role, as revealed by the integration of satiety and adiposity signalling.
Contrary to the traditional view of a hierarchical model in which the hypothalamus plays the major role, recent results are consistent with a distributed model in the processing of energy balance regulation. Interestingly, network re-organisation and neurogenesis are potentially involved in food intake and body weight regulation, suggesting that neuroplasticity may provide important clues to the understandingof energy balance disorders such as obesity.
Keys words : food intake, body weight, hypothalamus, brainstem, neuroplasticity.
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